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Betaglim (Glimepiride)
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Betaglim

Betaglim tells your pancreas to increase the amount of insulin it produces in your body. Insulin works to lower your blood sugar. Betaglim lowers your blood sugar in about 2 to 3 hours after you take the medication. This is why it is important to take the medication with your largest meal of the day.

Other names for this medication:
Asoride, Beglim, Biopride, Diaset, Forme, Geriglim, Glicon, Glimestar, Glimetis, Glimide, Glimilex, Glimiprime, Glimitab, Glimivir, Glimkap, Glimli, Glimpinor, Glipril, Glisen, Glypride, Glyrim, Glytab, Meprji, Metanorm, Metforise, Metride, Nabal, Nilconorm, Novaride, Pioglar, Piotop, Prichek, Vedapride, Vikpride, Voglow, Volpost, Zorep

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Also known as: Glimepiride

Description

{name} (glimepiride) is an oral diabetes medicine that helps control blood sugar levels.

{name} is used together with diet and exercise to improve blood sugar control in adults with type 2 diabetes mellitus. This medicine is not for treating type 1 diabetes.

Insulin or other diabetes medicines are sometimes used in combination with {name} if needed.

Dosage

Usual Adult Dose for Diabetes Type 2:

Initial dose: 1 to 2 mg orally once a day Maintenance dose: Increase in 1 or 2 mg increments no more frequently than every 1 to 2 weeks based on glycemic response Maximum dose: 8 mg per day Comments: -Administer with breakfast or first main meal of the day. -In patients who are at increased risk for hypoglycemia, start with 1 mg orally once a day and titrate slowly. -When patients are being transferred to this drug from a longer half-life sulfonylurea, monitor 1 to 2 weeks for overlapping drug effect. Use: As an adjunct to diet and exercise to improve glycemic control in patients with type 2 diabetes mellitus.

Usual Geriatric Dose for Diabetes Type 2:

Initial dose: 1 mg orally once a day; titrate slowly and monitor closely

Usual Pediatric Dose for Diabetes Type 2:

Not recommended because of adverse effects on body weight and hypoglycemia.

Detailed Betaglim dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. An Betaglim overdose can cause life-threatening hypoglycemia.

Symptoms of severe hypoglycemia include extreme weakness, confusion, tremors, sweating, fast heart rate, trouble speaking, nausea, vomiting, rapid breathing, fainting, and seizure (convulsions).

Storage

Store at room temperature below 30 degrees C (86 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Betaglim are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

You should not use Betaglim if you are allergic to glimepiride, sulphonamides, or if you have:

an allergy to sulfa drugs; or diabetic ketoacidosis (call your doctor for treatment).

To make sure Betaglim is safe for you, tell your doctor if you have ever had:

heart disease; liver or kidney disease; or an enzyme deficiency called glucose-6-phosphate dehydrogenase deficiency (G6PD).

Betaglim may increase your risk of serious heart problems, but not treating your diabetes can also damage your heart and other organs. Talk to your doctor about the risks and benefits of this medicine.

Follow your doctor's instructions about using Betaglim if you are pregnant or you become pregnant. Controlling diabetes is very important during pregnancy, and having high blood sugar may cause complications in both the mother and the baby. However, you may need to stop taking Betaglim for a short time just before your due date.

Medications similar to glimepiride have caused severe hypoglycemia in newborn babies whose mothers used the medicine near the time of delivery. If you take glimepiride during pregnancy, stop taking this medicine at least 2 weeks before your due date.

If you breastfeed while taking glimepiride, call your doctor if your baby shows signs of hypoglycemia (extreme drowsiness, feeding problems, mottled skin, blue lips, feeling cold or jittery, or having a seizure).

Betaglim is not approved for use by anyone younger than 18 years old.

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A significant increase of blood flow in the central cavernous arteries is the initial event leading to penile tumescence and rigidity. Fluorescently tagged iminoalditol glycosidase inhibitors as novel biological probes and diagnostics. Genome-wide analysis of DNA methylation in low cell numbers by reduced representation bisulfite sequencing. Membrane expansion of the erythrocyte by both the neutral and ionized forms of chlorpromazine. Gut suture and human amnion collagen more resembled living tissue than did bovine collagen and were characterized by their cellularity and the presence of numerous capillaries. Findings from two qualitative studies indicate the form of woman abuse to have differential impact on maternal response. The mechanisms underlying this panmucosal eosinophilia are unknown. Cardiac assist devices: present status and future opportunities. Chondroprotective and antiarthrotic properties of glycosaminoglycan polysulfate (GAGPS) in gonarthrosis in animal experiments.

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A European approach to rural-urban differences in mental health: the ESEMeD 2000 comparative study. It exhibits very high melting temperature and is thermally stable, shows superior single base mismatch discrimination capability, and is stable against digestion by nuclease and has no binding with single-stranded DNA binding proteins. Subsequently, on hemicraniotomy, the tip of the visor was found at the bifurcation of the internal carotid artery. Psychosocial correlates of blood pressure in childhood and adolescence. Pharmacological study of a quaternary ammonium compound possessing preponderant ganglionic stimulating properties. Recent investigations have revealed that erythrocytes from patients with chronic arterial occlusive disease are significantly less deformable than red blood cells from healthy subjects. We report a case of PCIS following cardiac surgery who showed false positive IgG, IgM antibodies to antigen A60 of Mycobacterium tuberculosis in pleural fluid. The myocardium showed fibre hypertrophy, interstitial fibrosis, vacuolar degeneration of myocardial cells and focal groups of aberrantly orientated myofibres.

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